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WAY-620147
CAS No. 515866-67-8
WAY-620147( —— )
Catalog No. M37287 CAS No. 515866-67-8
WAY-620147 is a derivative of N-(2-morpholinoethyl)nicotinamide, which is able to inhibit Monoamine Oxidase.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 29 | In Stock |
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| 10MG | 38 | In Stock |
|
| 25MG | 58 | In Stock |
|
| 50MG | 85 | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
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Product NameWAY-620147
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NoteResearch use only, not for human use.
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Brief DescriptionWAY-620147 is a derivative of N-(2-morpholinoethyl)nicotinamide, which is able to inhibit Monoamine Oxidase.
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DescriptionWAY-620147 (compound 6) is an N-(2-morpholinoethyl)nicotinamide derivative that inhibits monoamine oxidase (Monoamine Oxidase). WAY-620147 inhibits MAO-A and MAO-B with IC50s of 26 μM and 55 μM, respectively.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayMetabolic Enzyme/Protease
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TargetMAO
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RecptorMAO
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Research Area——
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Indication——
Chemical Information
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CAS Number515866-67-8
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Formula Weight314.18
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Molecular FormulaC12H16BrN3O2
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Purity>98% (HPLC)
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Solubility——
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SMILESO=C(NCCN1CCOCC1)C=2C=NC=C(Br)C2
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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AChE/BChE/MAO-B-IN-1
AChE/BChE/MAO-B-IN-1 is a reversible, non-time-dependent inhibitor of AChE, BChE, and MAO-B that crosses the blood-brain barrier, exhibiting inhibitory effects on hAChE, hBChE, and hMAO-B with IC50s of 7.31, 0.56, and 26.1 μM, respectively.
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Amiflamine
Amiflamine is an inhibitor of reversible MAO-A.The ex vivo approach was found to be valid for moclobemide in brain and liver and for cimoxatone in brain tissue;?a slight underestimation of the MAO A inhibitory effect of the latter in the liver is likely.?
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7,8-Dihydroxy-4-meth...
4-Methyldaphnetin is a potent inhibitor (low micromolar) of lipid peroxidation and scavengers of superoxide anion radicals and of aqueous alkylperoxyl radicals, but may be pro-oxidant (enhancing generation of hydroxyl radicals) in the presence of free iron ions.
