GQ-16

CAS No. 870554-67-9

GQ-16( —— )

Catalog No. M27488 CAS No. 870554-67-9

GQ-16 is a partial agonist of PPARγ with a Ki of 160 nM. GQ-16 reduces adipogenic actions and promotes insulin Sensitization without weight gain.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
1 mL x 10 mM in DMSO 29 In Stock
10MG 31 In Stock
25MG 69 In Stock
50MG 110 In Stock
100MG 182 In Stock
200MG 274 In Stock
500MG 440 In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    GQ-16
  • Note
    Research use only, not for human use.
  • Brief Description
    GQ-16 is a partial agonist of PPARγ with a Ki of 160 nM. GQ-16 reduces adipogenic actions and promotes insulin Sensitization without weight gain.
  • Description
    GQ-16 is a partial agonist of PPARγ with a Ki of 160 nM. GQ-16 reduces adipogenic actions and promotes insulin Sensitization without weight gain.
  • In Vitro
    ——
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Metabolic Enzyme/Protease
  • Target
    PPAR
  • Recptor
    Syk
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    870554-67-9
  • Formula Weight
    418.3
  • Molecular Formula
    C19H16BrNO3S
  • Purity
    >98% (HPLC)
  • Solubility
    In Vitro:?DMSO : 10 mg/mL (23.91 mM)
  • SMILES
    Cc1ccc(CN(C(/C(/S2)=C/c(cc(cc3)Br)c3OC)=O)C2=O)cc1
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1.International Nonproprietary Names for Pharmaceutical Substances (INN). WHO Drug Information, Vol. 31, No. 4, 2017.
molnova catalog
related products
  • Fenofibrate

    A PPARα agonist that exhibits EC50 of 18 and 30 uM for murine and human PPARα, respectively.

  • Dihydrocurcumin

    Dihydrocurcumin is the main metabolite of curcumin, which can reduce fat accumulation and oxidative stress response.

  • SR 16832

    SR 16832 is a dual-site PPARγ inhibitor. It also inhibits the binding of endogenous ligands and transcriptional activity of PPARγ, more effectively than the orthosteric covalent antagonist GW 9662 and T 0070907.