Desacetyl Forskolin

CAS No. 64657-20-1

Desacetyl Forskolin( ——— )

Catalog No. M39054 CAS No. 64657-20-1

Deacetylforskolin is a deacetylated derivative of forskolin that can be extracted from C. forskohlii. Deacetylforskolin activates rat adipocyte adenylyl cyclase (IC50 = 20 μM), inhibits glucose transport in rat adipocyte plasma membranes and exhibits antihypertensive activity.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
2MG 178 Get Quote
25MG Get Quote Get Quote
50MG Get Quote Get Quote
100MG Get Quote Get Quote
200MG Get Quote Get Quote
500MG Get Quote Get Quote
1G Get Quote Get Quote

Biological Information

  • Product Name
    Desacetyl Forskolin
  • Note
    Research use only, not for human use.
  • Brief Description
    Deacetylforskolin is a deacetylated derivative of forskolin that can be extracted from C. forskohlii. Deacetylforskolin activates rat adipocyte adenylyl cyclase (IC50 = 20 μM), inhibits glucose transport in rat adipocyte plasma membranes and exhibits antihypertensive activity.
  • Description
    Deacetylforskolin is a deacetylated derivative of forskolin that can be extracted from C. forskohlii. Deacetylforskolin activates rat adipocyte adenylyl cyclase (IC50 = 20 μM), inhibits glucose transport in rat adipocyte plasma membranes and exhibits antihypertensive activity.
  • In Vitro
    ———
  • In Vivo
    ———
  • Synonyms
    ———
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    ———
  • Research Area
    ———
  • Indication
    ———

Chemical Information

  • CAS Number
    64657-20-1
  • Formula Weight
    368.47
  • Molecular Formula
    C20H32O6
  • Purity
    >98% (HPLC)
  • Solubility
    ———
  • SMILES
    ———
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Joost HG, et al. Activation of adenylate cyclase and inhibition of glucose transport in rat adipocytes by forskolin analogues: structural determinants for distinct sites of action. Mol Pharmacol. 1988 Apr;33(4):449-53.?
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