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ZPCK
CAS No. 26049-94-5
ZPCK( Z-L-Phe chloromethyl ketone | NSC 251810 | SL 01 )
Catalog No. M13792 CAS No. 26049-94-5
ZPCK is a potent, pan autophagy inhibitor, inhibits the cargo degradation inside the vacuole and alters the process of autophagy in mammalian cells.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 873 | Get Quote |
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| 100MG | Get Quote | Get Quote |
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| 200MG | Get Quote | Get Quote |
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| 500MG | Get Quote | Get Quote |
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| 1G | Get Quote | Get Quote |
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Biological Information
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Product NameZPCK
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NoteResearch use only, not for human use.
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Brief DescriptionZPCK is a potent, pan autophagy inhibitor, inhibits the cargo degradation inside the vacuole and alters the process of autophagy in mammalian cells.
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DescriptionZPCK (NSC 251810;SL 01) is a potent, pan autophagy inhibitor, inhibits the cargo degradation inside the vacuole and alters the process of autophagy in mammalian cells; also modulates autophagic flux in a novel model plant, Aponogeton madagascariensis.
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In Vitro——
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In Vivo——
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SynonymsZ-L-Phe chloromethyl ketone | NSC 251810 | SL 01
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PathwayAutophagy
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TargetAutophagy
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RecptorAutophagy
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Research Area——
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Indication——
Chemical Information
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CAS Number26049-94-5
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Formula Weight331.796
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Molecular FormulaC18H18ClNO3
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Purity>98% (HPLC)
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Solubility——
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SMILESO=C(OCC1=CC=CC=C1)N[C@H](C(CCl)=O)CC2=CC=CC=C2
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Chemical NameN-[(1S)-3-chloro-2-oxo-1-(phenylmethyl)propyl]-carbamic acid, phenylmethyl ester
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Corynoxine hydrochlo...
Corynoxine is a natural oxindole alkaloid and induces autophagy in different neuronal cell lines including N2a and SHSY-5Y cells.
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LYN-1604 is a novel ULK1 activator.It inducing cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.?LYN-1604, to be the best candidate for a ULK1 agonist.?LYN-1604 induced cell death involved in ATF3, RAD21, and caspase3, accompanied by autophagy and apoptosis.?LYN-1604 has potential for good therapeutic effects on TNBC by targeting ULK1-modulated cell death in vivo.
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CP-312
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