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Sulmazole

CAS No. 73384-60-8

Sulmazole( —— )

Catalog No. M34433 CAS No. 73384-60-8

Sulmazole (AR-L 115-BS) is a small molecule Sulmazole is a small molecule cGMP-PDE inhibitor.Sulmazole is a competitive inhibitor of A1 adenosine receptors.

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
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Biological Information

  • Product Name
    Sulmazole
  • Note
    Research use only, not for human use.
  • Brief Description
    Sulmazole (AR-L 115-BS) is a small molecule Sulmazole is a small molecule cGMP-PDE inhibitor.Sulmazole is a competitive inhibitor of A1 adenosine receptors.
  • Description
    Sulmazole is a cardiotonic agent. Sulmazole has competitive inhibitory for A1 adenosine receptor. Sulmazole can improve cardiac index and reduce pulmonary capillary wedge pressure.
  • In Vitro
    Sulmazole has competitive inhibitory for A1 adenosine receptors.Sulmazole attenuates the capacity of GTP to inhibit adenylate cyclase activity and covalent modification of Gi by pertussis toxin treatment abolishes the capacity of sulmazole to mediate stimulation.Sulmazole has stimulatory effect on adenylate cyclase activity in pertussis toxin-treated membranes.Sulmazole mediates increases in cAMP accumulation by mechanisms other than phosphodiesterase inhibition, including A1 adenosine receptor antagonism and inhibition of Gi function.
  • In Vivo
    ——
  • Synonyms
    ——
  • Pathway
    Microbiology/Virology
  • Target
    Parasite
  • Recptor
    Parasite | Adenosine Receptor | PDE
  • Research Area
    ——
  • Indication
    ——

Chemical Information

  • CAS Number
    73384-60-8
  • Formula Weight
    287.34
  • Molecular Formula
    C14H13N3O2S
  • Purity
    >98% (HPLC)
  • Solubility
    ——
  • SMILES
    O(C)C1=C(C=CC(S(C)=O)=C1)C=2NC=3C(N2)=NC=CC3
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. W J Parsons, et al. The new cardiotonic agent sulmazole is an A1 adenosine receptor antagonist and functionally blocks the inhibitory regulator, Gi. Mol Pharmacol. 1988 Apr;33(4):441-8.?
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