Regaloside E

CAS No. 123134-21-4

Regaloside E( ——— )

Catalog No. M38403 CAS No. 123134-21-4

Regaloside E is a natural product that can be isolated from Lilium longiflorum Thunb. .

Purity : >98% (HPLC)

COA Datasheet HNMR HPLC MSDS Handing Instructions
Size Price / USD Stock Quantity
25MG Get Quote In Stock
50MG Get Quote In Stock
100MG Get Quote In Stock
200MG Get Quote In Stock
500MG Get Quote In Stock
1G Get Quote In Stock

Biological Information

  • Product Name
    Regaloside E
  • Note
    Research use only, not for human use.
  • Brief Description
    Regaloside E is a natural product that can be isolated from Lilium longiflorum Thunb. .
  • Description
    Regaloside E is a natural product that can be isolated from Lilium longiflorum Thunb.
  • In Vitro
    ———
  • In Vivo
    ———
  • Synonyms
    ———
  • Pathway
    Others
  • Target
    Other Targets
  • Recptor
    ———
  • Research Area
    ———
  • Indication
    ———

Chemical Information

  • CAS Number
    123134-21-4
  • Formula Weight
    458.416
  • Molecular Formula
    C20H26O12
  • Purity
    >98% (HPLC)
  • Solubility
    DMSO : ≥ 50 mg/mL (109.07 mM )
  • SMILES
    ———
  • Chemical Name
    ——

Shipping & Storage Information

  • Storage
    (-20℃)
  • Shipping
    With Ice Pack
  • Stability
    ≥ 2 years

Reference

1. Munafo JP Jr, et al. Quantitative Analysis of Phenylpropanoid Glycerol Glucosides in Different Organs of Easter Lily (Lilium longiflorum Thunb.). J Agric Food Chem. 2015 May 20;63(19):4836-42.?
molnova catalog
related products
  • Gymnoside I

    Gymnoside I (compound 1) is a glycosidoxybenzyl 2-isobutylmalic acid. Gymnoside I shows antiallergic activity in passive cutaneous allergic reaction (PCA) in mice. Gymnoside I can be used in research to treat asthma, neurasthenia and chronic hepatitis.

  • Homoarbutin

    Homoarbutin is a natural product.

  • Soyasapogenol A

    Soyasapogenol A shows estrogenic and hepatoprotective activities, it directly prevents apoptosis of hepatocytes, and secondly, inhibits the elevation of plasma TNF-α±, which consequently resulted in the prevention of liver damage in the concanavalin A-induced hepatitis model.