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3-O-α-L-Rhamnopyranosyl (1-2)[β-D-glucopyranosyl (1-4)]-α-L-arabinopyranosyl-27-hydroxyoleanolic acid 28-O-α-L-rhamnopyranosyl (1-4)-β-D-glucopyranosyl (1-6)-β-D-glucopyranoside
3-O-α-L-Rhamnopyranosyl (1-2)[β-D-glucopyranosyl (1-4)]-α-L-arabinopyranosyl-27-hydroxyoleanolic acid 28-O-α-L-rhamnopyranosyl (1-4)-β-D-glucopyranosyl (1-6)-β-D-glucopyranoside
CAS No. 938160-21-5
3-O-α-L-Rhamnopyranosyl (1-2)[β-D-glucopyranosyl (1-4)]-α-L-arabinopyranosyl-27-hydroxyoleanolic acid 28-O-α-L-rhamnopyranosyl (1-4)-β-D-glucopyranosyl (1-6)-β-D-glucopyranoside( ——— )
Catalog No. M38167 CAS No. 938160-21-5
Antitumor agent-89 (Compd OTS-5), a natural compound, weakly suppresses fMLP-induced superoxide generation.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
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Biological Information
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Product Name3-O-α-L-Rhamnopyranosyl (1-2)[β-D-glucopyranosyl (1-4)]-α-L-arabinopyranosyl-27-hydroxyoleanolic acid 28-O-α-L-rhamnopyranosyl (1-4)-β-D-glucopyranosyl (1-6)-β-D-glucopyranoside
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NoteResearch use only, not for human use.
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Brief DescriptionAntitumor agent-89 (Compd OTS-5), a natural compound, weakly suppresses fMLP-induced superoxide generation.
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DescriptionAntitumor agent-89 (Compd OTS-5), a natural compound, weakly suppresses fMLP-induced superoxide generation.
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In Vitro———
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In Vivo———
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Synonyms———
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PathwayOthers
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TargetOther Targets
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Recptor———
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Research Area———
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Indication———
Chemical Information
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CAS Number938160-21-5
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Formula Weight1383.52
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Molecular FormulaC65H106O31
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Purity>98% (HPLC)
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Solubility———
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SMILES———
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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GDC-1971
Migoprotafib (GDC-1971) (compound 199) is a SHP2 inhibitor. Migoprotafib inhibits the MAPK/ERK signaling pathway, and exhibits antitumor activity.
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2,3,4,5-TETRACHLOROP...
2,3,4,5-Tetrachlorophenol is a metabolite of γ-Lindane, which is an insecticide.
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Divalproex Sodium
Divalproex Sodium binds to and inhibits gamma-aminobutyric acid (GABA) transaminase and its anticonvulsant activity may be exerted by increasing brain concentration of GABA and by inhibiting enzymes that catabolize GABA or block the reuptake of GABA into glia and nerve endings.
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