Lecimibide
CAS No. 130804-35-2
Lecimibide( —— )
Catalog No. M34090 CAS No. 130804-35-2
Lecimibide (DuP 128) is a potent and selective inhibitor of acyl coenzyme A:cholesterol acyltransferase (ACAT), useful for studying high-lipid-related diseases.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 2MG | 222 | Get Quote |
|
| 5MG | 333 | Get Quote |
|
| 10MG | 504 | Get Quote |
|
| 25MG | 790 | Get Quote |
|
| 50MG | 1086 | Get Quote |
|
| 100MG | 1431 | Get Quote |
|
| 500MG | 2871 | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
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Product NameLecimibide
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NoteResearch use only, not for human use.
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Brief DescriptionLecimibide (DuP 128) is a potent and selective inhibitor of acyl coenzyme A:cholesterol acyltransferase (ACAT), useful for studying high-lipid-related diseases.
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DescriptionLecimibide (DuP 128) is a potent and specific acyl-CoA:cholesterol acyltransferase (ACAT) inhibitor for antihyperlipidemia research.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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RecptorAcyltransferase
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Research Area——
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Indication——
Chemical Information
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CAS Number130804-35-2
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Formula Weight590.77
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Molecular FormulaC34H40F2N4OS
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Purity>98% (HPLC)
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Solubility——
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SMILESS(CCCCCN(C(NC1=C(F)C=C(F)C=C1)=O)CCCCCCC)C=2NC(=C(N2)C3=CC=CC=C3)C4=CC=CC=C4
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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ACTH 1-14
ACTH (1-14) is a fragment of adrenocorticotrophin, which regulates cortisol and androgen production.Adrenocorticotropic hormone (ACTH), also known as corticotropin, is produced and secreted by the anterior pituitary gland. ACTH is an important component of the hypothalamic-pituitary-adrenal axis as a response to biological stress.
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Torachrysone gentiob...
Torachrysone gentiobioside is a phenolic glycoside isolated from cassia seeds.
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HIF-1 alpha 556-574
HIF-1 alpha (556-574) is a hypoxia-inducible factor-1 19-mer fragment. HIF-1 functions as master regulator of response to oxygen homeostasis. Hypoxia-induced gene expression is initiated when HIF-1subunit is stabilized in response to a lack of oxygen. This part of HIF-1 binds to the von Hippel-Lindau factor (VHL) an E3 ubiquitin ligase, and the proline 564 is absolutely vital to the binding process.
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