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Anhydrosafflor yellow B
CAS No. 184840-84-4
Anhydrosafflor yellow B( —— )
Catalog No. M29399 CAS No. 184840-84-4
Anhydrosafflor yellow B, isolated from Carthamus tinctorius, inhibits ADP-induced platelet aggregation, exhibits significant anti-oxidative effects in vitro, and possesses certain activity against H2O2-induced cytotoxicity.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 303 | Get Quote |
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| 100MG | Get Quote | Get Quote |
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| 200MG | Get Quote | Get Quote |
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| 500MG | Get Quote | Get Quote |
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| 1G | Get Quote | Get Quote |
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Biological Information
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Product NameAnhydrosafflor yellow B
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NoteResearch use only, not for human use.
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Brief DescriptionAnhydrosafflor yellow B, isolated from Carthamus tinctorius, inhibits ADP-induced platelet aggregation, exhibits significant anti-oxidative effects in vitro, and possesses certain activity against H2O2-induced cytotoxicity.
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DescriptionAnhydrosafflor yellow B, isolated from Carthamus tinctorius, inhibits ADP-induced platelet aggregation, exhibits significant anti-oxidative effects in vitro, and possesses certain activity against H2O2-induced cytotoxicity.
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In Vitro——
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In Vivo——
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Synonyms——
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PathwayOthers
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TargetOther Targets
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Recptor——
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Research Area——
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Indication——
Chemical Information
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CAS Number184840-84-4
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Formula Weight1044.91088867188
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Molecular FormulaC48H52O26
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Purity>98% (HPLC)
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Solubility——
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SMILESOC[C@@H](O)[C@@H](O)[C@H](O)[C@H]1OC2=C([C@@H]1C1=C(O)C(O)([C@@H]3O[C@H](CO)[C@@H](O)[C@H](O)[C@H]3O)C(O)=C(C(=O)\C=C\c3ccc(O)cc3)C1=O)C(=O)C(C(=O)\C=C\c1ccc(O)cc1)=C(O)C2(O)[C@@H]1O[C@H](CO)[C@@H](O)[C@H](O)[C@H]1O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Algestone acetopheni...
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ML-180
ML-180 is a potent orphan nuclear receptor liver receptor homolog 1 (LRH-1; NR5A2) inverse agonist (IC50 of 3.7 μM) .ML-180 (0.5-5 μM; 24 hours) shows a significant inhibition of cyclin-D1 and cyclin-E1 expression in hepatic cells, but has little effect on repression in SK-OV-3 cells[2]. ML-180 (5 μM; 24 hours) leads to a rapid decrease of LRH-1 expression and efficiently represses endogenous LRH-1 signaling.
