Cart
sales@molnova.com
Cinchonain Ib
CAS No. 85022-69-1
Cinchonain Ib( —— )
Catalog No. M29353 CAS No. 85022-69-1
Cinchonain Ib is isolated from Eriobotrya japonica leaves. Cinchonain Ib has an insulinotropic effect and can be used for stidies about managing type 2 diabetes.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 100MG | Get Quote | Get Quote |
|
| 200MG | Get Quote | Get Quote |
|
| 500MG | Get Quote | Get Quote |
|
| 1G | Get Quote | Get Quote |
|
Biological Information
-
Product NameCinchonain Ib
-
NoteResearch use only, not for human use.
-
Brief DescriptionCinchonain Ib is isolated from Eriobotrya japonica leaves. Cinchonain Ib has an insulinotropic effect and can be used for stidies about managing type 2 diabetes.
-
DescriptionCinchonain Ib is isolated from Eriobotrya japonica leaves. Cinchonain Ib has an insulinotropic effect and can be used for stidies about managing type 2 diabetes.(In Vitro):Cinchonain Ib enhanced significantly (p<0.05) insulin secretion from INS-1 cells.(In Vivo):Cinchonain Ib enhanced significantly (150%: p<0.05) plasma insulin level in rats for as long as 240 min after 108 mg/kg oral administration but did not induce any change in blood glucose level.
-
In Vitro——
-
In Vivo——
-
Synonyms——
-
PathwayOthers
-
TargetOther Targets
-
Recptor——
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number85022-69-1
-
Formula Weight452.41
-
Molecular FormulaC24H20O9
-
Purity>98% (HPLC)
-
Solubility——
-
SMILESO[C@H](C1)[C@@H](c(cc2)cc(O)c2O)Oc2c1c(O)cc(OC(C1)=O)c2[C@@H]1c(cc1)cc(O)c1O
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
molnova catalog
related products
-
4-fluoro-N-(3-fluoro...
4-fluoro-N-(3-fluoro-4-methylphenyl)-3-{[(4-methoxyphenyl)methyl]sulfamoyl}benzamide is a compound used as a molecular building block.
-
Rutarensin
Rutarensin is a phenolic compound found in Ruta chalepensis cell culture.
-
Elgodipine
Elgodipine significantly reduced the incidence and severity of exercise-induced angina pectoris systemically and was able to inhibit vascular smooth muscle proliferation through a mechanism independent of the expression of the transcription factors c-fos and c-jun.
