Cart
sales@molnova.com
W-5 hydrochloride
CAS No. 61714-25-8
W-5 hydrochloride( W-5 HCl | W 5 (hydrochloride) | N-(6-Aminohexyl)-1-naphthalenesulfonamide HCl )
Catalog No. M27739 CAS No. 61714-25-8
calmodulin antagonist.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 5MG | 40 | In Stock |
|
| 10MG | 59 | In Stock |
|
| 25MG | 119 | In Stock |
|
| 50MG | 177 | In Stock |
|
| 100MG | 259 | In Stock |
|
| 200MG | 388 | In Stock |
|
| 500MG | Get Quote | In Stock |
|
| 1G | Get Quote | In Stock |
|
Biological Information
-
Product NameW-5 hydrochloride
-
NoteResearch use only, not for human use.
-
Brief Descriptioncalmodulin antagonist.
-
Descriptioncalmodulin antagonist.(In Vitro):W-5 hydrochloride inhibits the proliferation of HR and KF cells with IC50 of 6.4μM and 8.6μM, respectively.
-
In Vitro——
-
In Vivo——
-
SynonymsW-5 HCl | W 5 (hydrochloride) | N-(6-Aminohexyl)-1-naphthalenesulfonamide HCl
-
PathwayOthers
-
TargetOther Targets
-
Recptor——
-
Research Area——
-
Indication——
Chemical Information
-
CAS Number61714-25-8
-
Formula Weight342.88
-
Molecular FormulaC16H23ClN2O2S
-
Purity>98% (HPLC)
-
Solubility——
-
SMILESCl.NCCCCCCNS(=O)(=O)c1cccc2ccccc12
-
Chemical Name——
Shipping & Storage Information
-
Storage(-20℃)
-
ShippingWith Ice Pack
-
Stability≥ 2 years
Reference
1.Do?owy M, et al. Application of various methods to determine the lipophilicity parameters of the selected urea pesticides as predictors of their bioaccumulation. J Environ Sci Health B. 2014;49(10):730-7.
molnova catalog
related products
-
AMDE-1
AMDE-1 is a modulator of autophagy activity that acts by triggering autophagy in an Atg5-dependent manner, recruiting Atg16 to the pre-autophagosomal site and causing LC3 lipidation.
-
ZXH-3-26 B
ZXH-3-26 is a PROTAC composed of a Cereblon ligand, an E3 ubiquitin ligase, and a BRD4 ligand that can be used to study cancer.
-
AGX51
AGX51 is an antagonist of inhibitor of DNA binding (ID) proteins. AGX51 treatment lead to pan-Id degradation, cell cycle arrest, and cell death.
