Ondansetron HCl
CAS No. 99614-01-4
Ondansetron HCl( GR 38032 (hydrochloride) | SN 307 (hydrochloride) )
Catalog No. M19305 CAS No. 99614-01-4
Ondansetron is a serotonin 5-HT3 receptor antagonist, used to prevent nausea and vomiting caused by cancer chemotherapy, and radiation therapy.
Purity : >98% (HPLC)
COA
Datasheet
HNMR
HPLC
MSDS
Handing Instructions
| Size | Price / USD | Stock | Quantity |
| 1 mL x 10 mM in DMSO | 37 | In Stock |
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| 25MG | 35 | In Stock |
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| 50MG | 49 | In Stock |
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| 100MG | Get Quote | In Stock |
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| 200MG | Get Quote | In Stock |
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| 500MG | Get Quote | In Stock |
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| 1G | Get Quote | In Stock |
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Biological Information
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Product NameOndansetron HCl
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NoteResearch use only, not for human use.
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Brief DescriptionOndansetron is a serotonin 5-HT3 receptor antagonist, used to prevent nausea and vomiting caused by cancer chemotherapy, and radiation therapy.
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DescriptionOndansetron is a serotonin 5-HT3 receptor antagonist, used to prevent nausea and vomiting caused by cancer chemotherapy, and radiation therapy.
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In Vitro——
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In Vivo——
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SynonymsGR 38032 (hydrochloride) | SN 307 (hydrochloride)
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PathwayOthers
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TargetOther Targets
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Recptor5-HT3
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Research AreaCancer|Neurological Disease
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Indication——
Chemical Information
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CAS Number99614-01-4
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Formula Weight329.82
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Molecular FormulaC18H19N3O·HCl
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Purity>98% (HPLC)
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Solubility——
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SMILESCl.CN1C2=CC=CC=C2C2=C1CCC(CN1C=CN=C1C)C2=O
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Chemical Name——
Shipping & Storage Information
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Storage(-20℃)
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ShippingWith Ice Pack
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Stability≥ 2 years
Reference
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Quercetin 3-O-6-O-(E...
Antioxidant agent-18 (compound 5) is a flavonol glycoside with antioxidant activity isolated from Ginkgo biloba. Antioxidant agent-18 scavenges DPPH radicals (IC50: 15.8 μM) and reduces cytochrome c (IC50: 14.7 μM).
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PAR-3 (1-6) amide (m...
SFNGGP-NH2 is a biological active peptide. (PAR-3 is a high-affinity thrombin receptor. PAR-3 mRNA is expressed in the cutaneous mast cells of humans. Protease-Activated Receptors (PARs) have been studied for their roles in itch and their itch-associated response through histamine-dependent/independent pathways have been reported. PAR-3 has been shown not to induce itching alone but possibly in conjunction with PAR-4. Co-expression of PAR-3 and PAR-4 enhances thrombin action suggesting that PAR-3 alone does not mediate transmembrane signaling but instead functions as a cofactor to activate PAR-4.)
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